Summary of Mediators of Acute Inflammation
{"Image DocumentName": "Summary of Mediators of Acute Inflammation", "Image ImageDescription": "ACTION. Mediator. Source. Vascular Leakage. Chemotaxis Other. Histamine and serotonin. Mast cells, platelets. + - Bradykinin. Plasma substrate. Pain. C3a. Plasma protein via liver. Opsonic fragment (C3b) C5a. Macrophages. Leukocyte adhesion, activation. Prostaglandins. Mast cells, from membrane phospholipids. Potentiate other mediators. Vasodilatation, pain, fever. Leukotriene B4. Leukocytes. Leukotrienes. C4 D4 E4. Leukocytes, mast cells. Bronchoconstriction, vasoconstriction. Platelet Activating Factor (PAF) Bronchoconstriction, leukocyte priming. IL-1 and TNF. Macrophages, other. Acute-phase reactions, endothelial activation. Chemokines. Leukocytes, others. Leukocyte activation. Macrophages, endothelium. Vasodilatation, cytotoxicity.", "Image XResolution": "37", "Image YResolution": "37", "Image ResolutionUnit": "Pixels/Centimeter", "Image Software": "SlidePlayer", "Image YCbCrPositioning": "Centered"}